Several studies mention how CBD might reduce inflammation and pain, which are the telltale signs of hemorrhoids.
First, it’s important that you stay physically active and eat healthy to maintain regular bowel movements. If your diet includes high amounts of carbohydrates and food to which humans are rather adapted than physiologically predisposed, then adding high-fiber foods may come in handy.
CBD Oil for Inflammation and Pain
Common hemorrhoid treatments include non-steroidal anti-inflammatory drugs (NSAIDs) and painkillers such as acetaminophen. However, some individuals have adverse reactions to these compounds; others are afraid of the dangerous side effects, hence the recent interest in natural remedies, such as CBD oil.
If hemorrhoids do occur, it’s best to consult a physician and discuss your treatment options, including taking CBD oil. A consultation with a qualified doctor will help you not only determine the right dosage for your case of hemorrhoids but also avoid potential interactions with other medications.
However, studies have yet to determine the right topical CBD dosing for pain relief in affected areas.
The new parent explained how she suffered with piles following the birth of her baby until discovering CBD, in post titled ‘CBD Saved My Butt (literally)’.
The poster used an unscented lotion that had the CBD compound in it, and found what she called ‘sweet heavenly mercy lawdy oh sweet relief’.
One mum shared on Reddit that it had ‘saved her butt’ after she gave birth and suffered with severe hemorrhoids. From there, plenty more people have been saying the same thing.
It’s unclear why and how CBD appears to work this way, and it’s important to ensure you always speak to your doctor before trying anything new rather than simply relying on personal anecdotes.
In it, she said: ‘I had my first baby in July. 3-4 weeks post-partum, I develop excruciating hemorrhoids (they sure don’t tell you it could happen!).
Today, the theory of sliding anal canal lining is widely accepted. This proposes that hemorrhoids develop when the supporting tissues of the anal cushions disintegrate or deteriorate. Hemorrhoids are therefore the pathological term to describe the abnormal downward displacement of the anal cushions causing venous dilatation. There are typically three major anal cushions, located in the right anterior, right posterior and left lateral aspect of the anal canal, and various numbers of minor cushions lying between them. The anal cushions of patients with hemorrhoids show significant pathological changes. These changes include abnormal venous dilatation, vascular thrombosis, degenerative process in the collagen fibers and fibroelastic tissues, distortion and rupture of the anal subepithelial muscle. In addition to the above findings, a severe inflammatory reaction involving the vascular wall and surrounding connective tissue has been demonstrated in hemorrhoidal specimens, with associated mucosal ulceration, ischemia, and thrombosis.
Recently, increased microvascular density was found in hemorrhoidal tissue, suggesting that neovascularization might be another important phenomenon of hemorrhoidal disease. Moreover, these workers found that microvascular density increased in hemorrhoidal tissue especially when thrombosis and stromal vascular endothelial growth factors (VEGF) were present.
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The exact pathophysiology of hemorrhoidal development is poorly understood. For years the theory of varicose veins, which postulated that hemorrhoids were caused by varicose veins in the anal canal, had been popular but now it is obsolete because hemorrhoids and anorectal varices are proven to be distinct entities. In fact, patients with portal hypertension and varices do not have an increased incidence of hemorrhoids.
Several enzymes or mediators involving the degradation of supporting tissues in the anal cushions have been studied. Among these, matrix metalloproteinase (MMP), a zinc-dependent proteinase, is one of the most potent enzymes, being capable of degrading extracellular proteins such as elastin, fibronectin, and collagen. MMP-9 was found to be over-expressed in hemorrhoids, in association with the breakdown of elastic fibers. Activation of MMP-2 and MMP-9 by thrombin, plasmin, or other proteinases resulted in the disruption of the capillary bed and promotion of angioproliferative activity of transforming growth factor β (TGF-β).
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